1 1775 72 PRACTICE OF YOGA MAY CAUSE DAMAGE OF BOTH SCIATIC NERVES: A CASE REPORT. SCIATIC NERVE TRAUMATIC DAMAGE VERY RARELY OCCURS BILATERALLY. WE DESCRIBE THE CASE OF A 67-YEAR-OLD WOMAN WHO REPORTED A BILATERAL TRAUMATIC LESION OF THE SCIATIC NERVE DURING PRACTICE OF YOGA. NERVE CONDUCTION STUDIES SHOWED A BILATERAL SCIATIC NERVE NEUROPATHY, MOSTLY AFFECTING THE PERONEAL COMPONENT. LUMBAR PLEXUS MRI DOCUMENTED REGULAR ANATOMICAL FEATURES OF THE MAIN PRINCIPAL NERVE ROOTS WITH BILATERAL T2 SIGNAL ALTERATION OF ROOTS L4, L5 AND S1 THAT EXTENDED INTO THE SCIATIC NERVES SHOWING BOTH INCREASE IN SIZE, PROBABLY RELATED TO CHRONIC INJURY OF NERVES, AND AN ALTERATION IN DIFFUSION SIGNAL THAT SUGGESTED A RECENT ACUTE OVERLAPPED PROCESS. 2013 2 1089 16 EFFECTS OF YOGA ON THE AUTONOMIC NERVOUS SYSTEM, GAMMA-AMINOBUTYRIC-ACID, AND ALLOSTASIS IN EPILEPSY, DEPRESSION, AND POST-TRAUMATIC STRESS DISORDER. A THEORY IS PROPOSED TO EXPLAIN THE BENEFITS OF YOGA PRACTICES IN DIVERSE, FREQUENTLY COMORBID MEDICAL CONDITIONS BASED ON THE CONCEPT THAT YOGA PRACTICES REDUCE ALLOSTATIC LOAD IN STRESS RESPONSE SYSTEMS SUCH THAT OPTIMAL HOMEOSTASIS IS RESTORED. IT IS HYPOTHESIZED THAT STRESS INDUCES (1) IMBALANCE OF THE AUTONOMIC NERVOUS SYSTEM (ANS) WITH DECREASED PARASYMPATHETIC NERVOUS SYSTEM (PNS) AND INCREASED SYMPATHETIC NERVOUS SYSTEM (SNS) ACTIVITY, (2) UNDERACTIVITY OF THE GAMMA AMINO-BUTYRIC ACID (GABA) SYSTEM, THE PRIMARY INHIBITORY NEUROTRANSMITTER SYSTEM, AND (3) INCREASED ALLOSTATIC LOAD. IT IS FURTHER HYPOTHESIZED THAT YOGA-BASED PRACTICES (4) CORRECT UNDERACTIVITY OF THE PNS AND GABA SYSTEMS IN PART THROUGH STIMULATION OF THE VAGUS NERVES, THE MAIN PERIPHERAL PATHWAY OF THE PNS, AND (5) REDUCE ALLOSTATIC LOAD. DEPRESSION, EPILEPSY, POST TRAUMATIC STRESS DISORDER (PTSD), AND CHRONIC PAIN EXEMPLIFY MEDICAL CONDITIONS THAT ARE EXACERBATED BY STRESS, HAVE LOW HEART RATE VARIABILITY (HRV) AND LOW GABAERGIC ACTIVITY, RESPOND TO PHARMACOLOGIC AGENTS THAT INCREASE ACTIVITY OF THE GABA SYSTEM, AND SHOW SYMPTOM IMPROVEMENT IN RESPONSE TO YOGA-BASED INTERVENTIONS. THE OBSERVATION THAT TREATMENT RESISTANT CASES OF EPILEPSY AND DEPRESSION RESPOND TO VAGAL NERVE STIMULATION CORROBORATES THE NEED TO CORRECT PNS UNDERACTIVITY AS PART OF A SUCCESSFUL TREATMENT PLAN IN SOME CASES. ACCORDING TO THE PROPOSED THEORY, THE DECREASED PNS AND GABAERGIC ACTIVITY THAT UNDERLIES STRESS-RELATED DISORDERS CAN BE CORRECTED BY YOGA PRACTICES RESULTING IN AMELIORATION OF DISEASE SYMPTOMS. THIS HAS FAR-REACHING IMPLICATIONS FOR THE INTEGRATION OF YOGA-BASED PRACTICES IN THE TREATMENT OF A BROAD ARRAY OF DISORDERS EXACERBATED BY STRESS. 2012 3 781 13 EFFECT OF YOGA ASANAS ON NERVE CONDUCTION IN TYPE 2 DIABETES. TWENTY TYPE 2 DIABETIC SUBJECTS BETWEEN THE AGE GROUP OF 30-60 YEARS WERE STUDIED TO SEE THE EFFECT OF 40 DAYS OF YOGA ASANAS ON THE NERVE CONDUCTION VELOCITY. THE DURATION OF DIABETES RANGED FROM 0-10 YEARS. SUBJECT SUFFERING FROM CARDIAC, RENAL AND PROLIFERATIVE RETINAL COMPLICATIONS WERE EXCLUDED FROM THE STUDY YOGA ASANAS INCLUDED SURYANAMSKAR. TADASAN, KONASAN, PADMASAN PRANAYAM, PASCHIMOTTANSAN ARDHMATSYENDRASAN, SHAVASAN, PAVANMUKTHASAN, SARPASAN AND SHAVASAN. SUBJECTS WERE CALLED TO THE CARDIO-RESPIRATORY LABORATORY IN THE MORNING TIME AND WERE GIVEN TRAINING BY THE YOGA EXPERT. THE YOGA EXERCISES WERE PERFORMED FOR 30-40 MINUTES EVERY DAY FOR 40 DAYS IN THE ABOVE SEQUENCE. THE SUBJECTS WERE PRESCRIBED CERTAIN MEDICINES AND DIET. THE BASAL BLOOD GLUCOSE, NERVE CONDUCTION VELOCITY OF THE MEDIAN NERVE WAS MEASURED AND REPEATED AFTER 40 DAYS OF YOGIC REGIME. ANOTHER GROUP OF 20 TYPE 2 DIABETES SUBJECTS OF COMPARABLE AGE AND SEVERITY, CALLED THE CONTROL GROUP, WERE KEPT ON PRESCRIBED MEDICATION AND LIGHT PHYSICAL EXERCISES LIKE WALKING. THEIR BASAL & POST 40 DAYS PARAMETERS WERE RECORDED FOR COMPARISON. RIGHT HAND AND LEFT HAND MEDIAN NERVE CONDUCTION VELOCITY INCREASED FROM 52.81 +/- 1.1 M/SEC TO 53.87 +/- 1.1 M/SEC AND 52.46 +/- 1.0 TO 54.75 +/- 1/1 M/SEC RESPECTIVELY. CONTROL GROUP NERVE FUNCTION PARAMETERS DETERIORATED OVER THE PERIOD OF STUDY, INDICATING THAT DIABETES IS A SLOWLY PROGRESSIVE DISEASE INVOLVING THE NERVES. YOGA ASANAS HAVE A BENEFICIAL EFFECT ON GLYCAEMIC CONTROL AND IMPROVE NERVE FUNCTION IN MILD TO MODERATE TYPE 2 DIABETES WITH SUB-CLINICAL NEUROPATHY. 2002 4 2724 15 YOGA NEUROPATHY. A SNOOZER. SCIATIC NERVE COMPRESSION VERY RARELY OCCURS BILATERALLY. THE AUTHORS PRESENT A WOMAN WITH PROFOUND LOWER EXTREMITY WEAKNESS AND SENSORY ABNORMALITY AFTER FALLING ASLEEP IN THE HEAD-TO-KNEES YOGA POSITION (ALSO CALLED "PASCHIMOTTANASANA"). CLINICAL AND ELECTRODIAGNOSTIC FINDINGS ARE DISCUSSED IN DETAIL AND A BRIEF REVIEW OF THE LITERATURE IS PRESENTED. 2005 5 2832 16 YOGA'S BIOPHYSIOLOGICAL EFFECTS ON LOWER URINARY TRACT SYMPTOMS: A SCOPING REVIEW. BACKGROUND AND OBJECTIVES: YOGA IS A MIND AND BODY PRACTICE THAT INCLUDES RELAXATION, MEDITATION, BREATHING EXERCISES, AND BODY POSTURES. IT CAN BE EFFECTIVE IN ENHANCING THE FUNCTIONING OF SEVERAL BODY SYSTEMS, INCLUDING THE LOWER URINARY TRACT. NORMAL LOWER URINARY TRACT FUNCTIONING DEPENDS IN PART ON THE COORDINATION OF THE BLADDER, URETHRA, PELVIC FLOOR AND OTHER MUSCLES, AND THE NERVES THAT CONTROL THEM. LOWER URINARY TRACT DYSFUNCTION CAN LEAD TO SYMPTOMS, THAT IS, STRESS URINARY INCONTINENCE (UI), URINARY FREQUENCY, NOCTURIA, URINARY URGENCY WITH AND WITHOUT INCONTINENCE, AND MIXED UI. RECENT EVIDENCE SUGGESTS THAT YOGA CAN IMPROVE LOWER URINARY TRACT SYMPTOMS (LUTS). THUS, WE PERFORMED A SCOPING REVIEW OF THE LITERATURE WITH REGARD TO THE EVIDENCE FOR THE EFFECTS OF YOGA ON LUTS AND FACTORS THAT MAY MEDIATE YOGA'S EFFECTS ON LUTS WITH THE GOAL TO IDENTIFY GAPS IN KNOWLEDGE REGARDING THE RELATIONSHIP BETWEEN YOGA PRACTICE AND LUTS. METHODS: THE AUTHORS EMPLOYED THE PRISMA EXTENSION FOR SCOPING REVIEWS (PRISMA-SCR) METHODOLOGICAL APPROACH, PROPOSED BY TRICCO ET AL., BY SEARCHING THE ELECTRONIC DATABASES, PUBMED, EMBASE, AND PSYCINFO, FOR ARTICLES USING THE FOLLOWING KEYWORDS: YOGA, URINARY INCONTINENCE, URINARY TRACT, BLADDER, AND URETHRA. WE ASSESSED THE QUALITY OF THE STUDIES USING THE JOANNA BRIGGS INSTITUTE CRITICAL APPRAISAL CHECKLIST. RESULTS: OF THE 172 ARTICLES WE FOUND, 8 ARTICLES MET THE INCLUSION CRITERIA AND WERE REVIEWED. WE FOUND THAT, DESPITE THE USE OF DIFFERENT PROTOCOLS, YOGA MAY REDUCE CERTAIN LUTS BY INCREASING THE STRENGTH OF PELVIC FLOOR MUSCLE AND/OR REGULATING THE AUTONOMIC NERVOUS SYSTEM AND ACTIVATING THE CENTRAL NERVOUS SYSTEM. CONCLUSIONS: YOGA IS A NONINVASIVE PRACTICE THAT MAY IMPROVE SOME LUTS. RIGOROUS STUDIES ARE NEEDED TO DETERMINE THE SPECIFIC MECHANISMS THROUGH WHICH YOGA MAY AFFECT LUTS. 2019 6 138 10 A PRELIMINARY REPORT ON THE ROLE OF YOGA ASANAS ON OXIDATIVE STRESS IN NON-INSULIN DEPENDENT DIABETES MELLITUS. NINETEEN SUBJECTS OF NON-INSULIN DEPENDENT DIABETES MELLITUS (NIDDM) BETWEEN THE AGE GROUP OF 30-60 YRS WERE STUDIED TO SEE THE EFFECT OF SPECIFIC YOGA ASANAS ON FASTING AND POSTPRANDIAL BLOOD GLUCOSE (FBG, PPG), SERUM MALONDIALDEHYDE (MDA) AND GLYCOSYLATED HEMOGLOBIN (HBA(1)) IN ADDITION TO DRUG TREATMENT AND DIET CONTROL. THE DURATION OF DIABETES RANGED FROM 1-10 YEARS. PATIENTS WITH RENAL, CARDIAC AND PROLIFERATIVE RETINAL DISEASES WERE EXCLUDED FROM THE STUDY. THE SAME PATIENTS SERVED AS THEIR OWN CONTROL. SUBJECTS WERE CALLED IN THE MORNING TO THE CARDIO-RESPIRATORY LABORATORY AND WERE GIVEN TRAINING BY A YOGA EXPERT. YOGA ASANAS INCLUDED SURYANAMSKAR, TADASAN, TRIKONASAN, PADMASAN, PRANAYAM, PASCHIMOTTANASAN, ARDHMATSYENDRASAN, PAVANMUKTHASAN, SARPASAN AND SHAVASAN. THE ASANAS WERE DONE EVERY DAY FOR 40 DAYS FOR 30-40 MIN. FBG, PPG, SERUM MDA AND HBA(1) WERE ESTIMATED BEFORE AND AFTER 40 DAYS OF YOGA ASANAS REGIMEN. SIGNIFICANT REDUCTION WAS SEEN IN FBG FROM 220 MG/DL TO 162 MG/DL, PPG FROM 311 MG/DL TO 255 MG/DL, MDA FROM 6 NMOL/L TO 3 NMOL/L AND HBA(1), FROM 8.8% TO 6.4%. SUBJECTS FELT BETTER AND WERE RELIEVED OF THEIR STRESSES AND HAD AN IMPROVEMENT IN THEIR DAY TO DAY PERFORMANCE. THE DECREASE WAS STATISTICALLY SIGNIFICANT (P<0.0001 FOR FBG AND PPG, P<0.001 FOR MDA AND FOR HBA(1)). 2001 7 2694 15 YOGA INDUCED ACUTE ULNAR NERVE COMPRESSION BY A GANGLION CYST IN GUYON'S CANAL. ACUTE ULNAR NEUROPATHY AT THE WRIST CAN BE DIFFICULT TO DIAGNOSE, AS IT IS AN UNCOMMON NEUROPATHY WITH VARIABLE CLINICAL PRESENTATIONS AND NUMEROUS ETIOLOGIES. WE PRESENT A CASE OF ACUTE ULNAR NEUROPATHY OF THE DEEP MOTOR BRANCH CAUSED BY A GANGLION CYST IN GUYON'S CANAL. INTERESTINGLY, THIS CASE OF ACUTE LOSS OF MOTOR FUNCTION OCCURRED AFTER THE PATIENT PARTICIPATED IN YOGA (SPECIFICALLY THE DOWNWARD DOG POSITION), AND RESOLVED SPONTANEOUSLY OVER TIME AFTER STOPPING YOGA, WITHOUT SURGICAL EXCISION OF THE GANGLION, SUGGESTING EXACERBATION OR PROTRUSION OF AN OCCULT GANGLION CYST DUE TO INCREASED ACTIVITY AND COMPRESSION OF THE HYPOTHENAR EMINENCE. 2013 8 887 8 EFFECT OF YOGA TYPE BREATHING ON HEART RATE AND CARDIAC AXIS OF NORMAL SUBJECTS. EFFECT OF INSPIRATORY AND EXPIRATORY PHASES OF NORMAL QUIET BREATHING, DEEP BREATHING AND SAVITRI PRANAYAM TYPE BREATHING ON HEART RATE AND MEAN VENTRICULAR QRS AXIS WAS INVESTIGATED IN YOUNG, HEALTHY UNTRAINED SUBJECTS. PRANAYAM TYPE BREATHING PRODUCED SIGNIFICANT CARDIOACCELERATION AND INCREASE IN QRS AXIS DURING THE INSPIRATORY PHASE AS COMPARED TO EUPNEA. ON THE OTHER HAND, EXPIRATORY EFFORT DURING PRANAYAM TYPE BREATHING DID NOT PRODUCE ANY SIGNIFICANT CHANGE IN HEART RATE OR QRS AXIS. THE CHANGES IN HEART RATE AND QRS AXIS DURING THE INSPIRATORY AND EXPIRATORY PHASES OF PRANAYAM TYPE BREATHING WERE SIMILAR TO THE CHANGES OBSERVED DURING THE CORRESPONDING PHASES OF DEEP BREATHING. 1986 9 2056 13 THE BENEFICIAL EFFECT OF YOGA IN DIABETES. TWENTY NIDDM SUBJECTS (MILD TO MODERATE DIABETICS) IN THE AGE GROUP OF 30-60 YEARS WERE SELECTED FROM THE OUT PATIENT CLINIC OF G.T.B. HOSPITAL. THEY WERE ON A 40 DAYS YOGA ASANA REGIME UNDER THE SUPERVISION OF A YOGA EXPERT. 13 SPECIFIC YOGA ASANAS < OR = DONE BY TYPE 2 DIABETES PATIENTS INCLUDED. SURYA NAMASKAR, TRIKONASANA, TADASANA, SUKHASANA, PADMASANA, BHASTRIKA PRANAYAMA, PASHIMOTTANASANA, ARDHMATSYENDRASANA, PAWANMUKTASANA, BHUJANGASANA, VAJRASANA, DHANURASANA AND SHAVASANA ARE BENEFICIAL FOR DIABETES MELLITUS. SERUM INSULIN, PLASMA FASTING AND ONE HOUR POSTPRANDIAL BLOOD GLUCOSE LEVELS AND ANTHROPOMETRIC PARAMETERS WERE MEASURED BEFORE AND AFTER YOGA ASANAS. THE RESULTS INDICATE THAT THERE WAS SIGNIFICANT DECREASE IN FASTING GLUCOSE LEVELS FROM BASAL 208.3 +/- 20.0 TO 171.7 +/- 19.5 MG/DL AND ONE HOUR POSTPRANDIAL BLOOD GLUCOSE LEVELS DECREASED FROM 295.3 +/- 22.0 TO 269.7 +/- 19.9 MG/DL. THE EXACT MECHANISM AS TO HOW THESE POSTURES AND CONTROLLED BREATHING INTERACT WITH SOMATOENDOCRINE MECHANISM AFFECTING INSULIN KINETICS WAS WORKED OUT. A SIGNIFICANT DECREASE IN WAIST-HIP RATIO AND CHANGES IN INSULIN LEVELS WERE ALSO OBSERVED, SUGGESTING A POSITIVE EFFECT OF YOGA ASANAS ON GLUCOSE UTILISATION AND FAT REDISTRIBUTION IN NIDDM. YOGA ASANAS MAY BE USED AS AN ADJUNCT WITH DIET AND DRUGS IN THE MANAGEMENT OF TYPE 2 DIABETES. 2005 10 2886 15 YOGA: BALANCING THE EXCITATION-INHIBITION EQUILIBRIUM IN PSYCHIATRIC DISORDERS. SOCIAL BEHAVIORAL DISTURBANCES ARE CENTRAL TO MOST PSYCHIATRIC DISORDERS. A DISEQUILIBRIUM WITHIN THE CORTICAL EXCITATORY AND INHIBITORY NEUROTRANSMITTER SYSTEMS UNDERLIES THESE DEFICITS. GAMMA-AMINOBUTYRIC ACID (GABA) AND GLUTAMATE ARE THE MOST ABUNDANT EXCITATORY AND INHIBITORY NEUROTRANSMITTERS IN THE BRAIN THAT CONTRIBUTE TO THIS EQUILIBRIUM. SEVERAL CONTEMPORARY THERAPIES USED IN TREATING PSYCHIATRIC DISORDERS, REGULATE THIS GABA-GLUTAMATE BALANCE. YOGA HAS BEEN STUDIED AS AN ADJUVANT TREATMENT ACROSS A BROAD RANGE OF PSYCHIATRIC DISORDERS AND IS SHOWN TO HAVE SHORT-TERM THERAPEUTIC GAINS. EMERGING EVIDENCE FROM RECENT CLINICAL IN VIVO EXPERIMENTS SUGGESTS THAT YOGA IMPROVES GABA-MEDIATED CORTICAL-INHIBITORY TONE AND ENHANCES PERIPHERAL OXYTOCIN LEVELS. THIS IS LIKELY TO HAVE A MORE CONTROLLED DOWNSTREAM RESPONSE OF THE HYPOTHALAMO-PITUITARY-ADRENAL SYSTEM BY MEANS OF REDUCED CORTISOL RELEASE AND HENCE A BLUNTED SYMPATHETIC RESPONSE TO STRESS. ANIMAL AND EARLY FETAL DEVELOPMENTAL STUDIES SUGGEST AN INTER-DEPENDENT ROLE OF OXYTOCIN AND GABA IN REGULATING SOCIAL BEHAVIORS. IN KEEPING WITH THESE OBSERVATIONS, WE PROPOSE AN INTEGRATED NEUROBIOLOGICAL MODEL TO STUDY THE MECHANISMS OF THERAPEUTIC BENEFITS WITH YOGA. APART FROM PROVIDING A NEUROSCIENTIFIC BASIS FOR APPLYING A TRADITIONAL SYSTEM OF PRACTICE IN THE CLINICAL SETTING, THIS MODEL CAN BE USED AS A FRAMEWORK FOR STUDYING YOGA MECHANISMS IN FUTURE CLINICAL TRIALS. 2019 11 1813 17 PROGRESSION OF GLAUCOMA ASSOCIATED WITH THE SIRSASANA (HEADSTAND) YOGA POSTURE. THIS ARTICLE REPORTS A CASE OF PROGRESSIVE GLAUCOMATOUS OPTIC NEUROPATHY AND VISUAL FIELD LOSS THAT OCCURRED IN A PATIENT WHO PRACTICED THE SIRSASANA (HEADSTAND) YOGA POSTURE ON A DAILY BASIS FOR MANY YEARS. VISUAL FIELD ANALYSIS WAS PERFORMED THROUGH STANDARD AUTOMATED PERIMETRY. INTRAOCULAR PRESSURE (IOP) WAS MEASURED THROUGH PNEUMOTONOMETRY IN THE SITTING POSITION AND IN THE HEAD-DOWN POSITION. STEREO-OPTIC DISC PHOTOGRAPHS WERE OBTAINED. IOP INCREASED SIGNIFICANTLY IN THE HEAD-DOWN POSITION. OPTIC DISC EVALUATION REVEALED A NEW DISC HEMORRHAGE IN THE LEFT EYE. VISUAL FIELD ANALYSIS OVER A PERIOD OF 2 Y SHOWED PROGRESSION OF A SUPERIOR ARCUATE DEFECT IN THE LEFT EYE. TRANSIENT INCREASES IN IOP ASSOCIATED WITH THE YOGA HEADSTAND POSTURE MAY LEAD TO PROGRESSIVE GLAUCOMATOUS OPTIC NERVE DAMAGE AND VISUAL FIELD LOSS. 2006 12 1952 9 SEASONAL AFFECTIVE DISORDER AND THE YOGA PARADIGM: A RECONSIDERATION OF THE ROLE OF THE PINEAL GLAND. SEASONAL AFFECTIVE DISORDER IS A PSYCHIATRIC DISORDER WHOSE PATHOPHYSIOLOGY AND CLINICAL PRESENTATION ARE POORLY UNDERSTOOD. BY APPLYING THE ANCIENT PARADIGM OF YOGA PSYCHOLOGY TO THIS SUBJECT, NEW UNDERSTANDINGS OF THE SYNDROME EMERGE REGARDING THE POSSIBLE ROLE OF THE PINEAL GLAND, THE CLINICAL PRESENTATION OF THE SYNDROME, AND THE POSSIBLE MECHANISM OF ACTION OF PHOTOTHERAPY. THE ENERGY DEPLETION MODEL PRESENTED HERE TIES TOGETHER SUCH DIVERSE ELEMENTS AS: DOSE-RESPONSE ASPECTS OF PHOTOTHERAPY, ANERGIA AS A PRIMARY SYMPTOM OF SAD, 'SPRING FEVER', MYOFASCIAL PAIN DISORDER, THE ANTI-GONADOTROPHIC EFFECT OF MELATONIN, AND PINEAL SUPERSENSITIVITY IN BIPOLAR PATIENTS. CLINICAL PREDICTIONS ARE MADE, AND SIMPLE RESEARCH PROTOCOLS ARE SUGGESTED WHICH CAN DIRECTLY TEST THE HYPOTHESES GENERATED BY THIS PARADIGM. 1990 13 264 16 ACUTE CHEST PAIN AND ESOPHAGEAL MUCOSAL INJURY FOLLOWING AN EXTREME YOGA POSITION CASE REPORT. A YOUNG LADY COMPLAINED OF THE SUDDEN ONSET OF INTENSE CHEST PAIN, IN CONSEQUENCE OF AN EXTREME HYPEREXTENSION OF THE BACK IN A YOGA POSITION. AT ENDOSCOPY A LARGE LESION OF THE ESOPHAGEAL EPITHELIUM WAS DETECTED, INVOLVING THE MIDDLE THIRD OF THE ANTERIOR WALL OF THE ESOPHAGUS. OTHER SYMPTOMS REPORTED BY THE PATIENT WERE DYSPHAGIA AND ODYNOPHAGIA, DEPICTING THE TYPICAL FEATURES OF INTRAMURAL HEMATOMA, ALSO KNOWN AS INTRAMURAL DISSECTION OR INTRAMURAL PERFORATION OF THE OESOPHAGUS. THE PATIENT WAS MANAGED CONSERVATIVELY AND SYMPTOMS DISAPPEARED WITHIN A WEEK. A BARIUM SWALLOW AT SIX MONTHS REPORTED NORMAL FINDINGS. DIFFERENT TYPES OF ACCIDENTS OCCURRING DURING YOGA PRACTICE ARE REPORTED IN THE LITERATURE, MAINLY INVOLVING MUSCULOSKELETAL OR NERVOUS SYSTEMS. VISCERAL LESIONS ARE EXCEPTIONAL AND NO SIMILAR CASES HAVE BEEN REPORTED IN THE LITERATURE. KEYWORDS: ACUTE CHEST PAIN, ESOPHAGEAL LESION, INTRAMURAL HEMATOMA, MANAGEMENT OF ESOPHAGEAL LESION. 2019 14 655 16 EEG PAROXYSMAL GAMMA WAVES DURING BHRAMARI PRANAYAMA: A YOGA BREATHING TECHNIQUE. HERE WE REPORT THAT A SPECIFIC FORM OF YOGA CAN GENERATE CONTROLLED HIGH-FREQUENCY GAMMA WAVES. FOR THE FIRST TIME, PAROXYSMAL GAMMA WAVES (PGW) WERE OBSERVED IN EIGHT SUBJECTS PRACTICING A YOGA TECHNIQUE OF BREATHING CONTROL CALLED BHRAMARI PRANAYAMA (BHPR). TO OBTAIN NEW INSIGHTS INTO THE NATURE OF THE EEG DURING BHPR, WE ANALYZED EEG SIGNALS USING TIME-FREQUENCY REPRESENTATIONS (TFR), INDEPENDENT COMPONENT ANALYSIS (ICA), AND EEG TOMOGRAPHY (LORETA). WE FOUND THAT THE PGW CONSISTS OF HIGH-FREQUENCY BIPHASIC RIPPLES. THIS UNUSUAL ACTIVITY IS DISCUSSED IN RELATION TO PREVIOUS REPORTS ON YOGA AND MEDITATION. IT IS CONCLUDED THIS EEG ACTIVITY IS MOST PROBABLY NON-EPILEPTIC, AND THAT APPLYING THE SAME METHODOLOGY TO OTHER MEDITATION RECORDINGS MIGHT YIELD AN IMPROVED UNDERSTANDING OF THE NEUROCORRELATES OF MEDITATION. 2009 15 433 9 CAN YOGA THERAPY STIMULATE STEM CELL TRAFFICKING FROM BONE MARROW? IT HAS BEEN ESTABLISHED THAT MESENCHYMAL STROMAL CELLS (MSCS) FROM BONE MARROW ENTER THE PERIPHERAL CIRCULATION INTERMITTENTLY FOR POSSIBLE TISSUE REGENERATION, REPAIR AND TO TAKE CARE OF DAILY WEAR AND TEAR. THIS IS EVIDENT FROM THE DETECTION OF MSCS FROM PERIPHERAL BLOOD. THE FACTORS GOVERNING THIS MIGRATION REMAIN ELUSIVE. THESE MSCS CARRY OUT THE WORK OF POLICING AND ARE SUPPOSED TO REPAIR THE INJURED TISSUES. THUS, THESE CELLS HELP IN MAINTAINING THE TISSUE AND ORGAN HOMEOSTASIS. YOGA AND PRANAYAMA ORIGINATED IN INDIA AND IS NOW BEING PRACTICED ALL OVER THE WORLD FOR POSITIVE HEALTH. SO FAR, THE CHEMICAL STIMULATION OF BONE MARROW HAS BEEN WIDELY USED EMPLOYING INJECTION OF COLONY STIMULATING FACTOR. HOWEVER, THE ROLE OF PHYSICAL FACTORS SUCH AS MECHANICAL STIMULATION AND STRETCHING HAS NOT BEEN SUBSTANTIATED. IT IS CLAIMED THAT PRACTICING YOGA DELAYS SENESCENCE, IMPROVES THE PHYSIOLOGICAL FUNCTIONS OF HEART AND LUNG AND YOGA POSTURES MAKE THE BODY ELASTIC. IT REMAINS TO BE SEEN WHETHER THE YOGA THERAPY PROMOTES TRAFFICKING OF THE STEM CELLS FROM BONE MARROW FOR POSSIBLE REPAIR AND REGENERATION OF WORN OUT AND DEGENERATING TISSUES. WE COVER IN THIS SHORT REVIEW, MAINLY THE ROLE OF PHYSICAL FACTORS ESPECIALLY THE YOGA THERAPY ON STEM CELLS TRAFFICKING FROM BONE MARROW. 2016 16 407 11 BIOLOGICAL SIGNIFICANCE OF PIEZOELECTRICITY IN RELATION TO ACUPUNCTURE, HATHA YOGA, OSTEOPATHIC MEDICINE AND ACTION OF AIR IONS. PIEZOELECTRIC PROPERTIES OF BIOLOGICAL MACROMOLECULES SUCH AS PROTEINS, NUCLEIC ACIDS AND MUCOPOLYSACCHARIDES ARE REVIEWED IN THIS PAPER. IT IS INDICATED THAT THE STRUCTURAL ELEMENTS OF THE HUMAN BODY COMPOSED OF THESE PIEZOELECTRIC SUBSTANCES ARE CAPABLE OF TRANSDUCING A MECHANICAL ENERGY INTO AN ELECTRIC CURRENT. SUCH A TRANSDUCTION MAY BE BROUGHT ABOUT BY MOVEMENTS OF AN ACUPUNCTURE NEEDLE, OSTEOPATHIC MANIPULATIONS; HATHA YOGA POSTURES OR ACTION OF NEGATIVELY CHARGED AIR IRONS. IT IS POSTULATED THAT ELECTRIC CURRENT INDUCED BY STIMULATION OF THE SPECIFIC SITES ON THE SURFACE OF HUMAN BODY FLOWS TOWARDS THE INTERNAL ORGANS ALONG THE SEMICONDUCTIVE CHANNELS OF BIOLOGIC MACROMOLECULES. ELECTRIC CURRENT INDUCED EITHER BY THE PIEZOELECTRIC TRANSDUCTION OR DIRECTLY APPLIED FROM AN EXTERNAL SOURCE MAY IN TURN STIMULATE INDIVIDUAL CELLS IN THE TARGET ORGAN. INVOLVEMENT OF ELECTRICAL PHENOMENA IN REGULATORY MECHANISMS ON CELLULAR AND MOLECULAR LEVELS IS DISCUSSED. 1977 17 2779 11 YOGA TECHNIQUES AS A MEANS OF CORE STABILITY TRAINING. CORE STABILITY IN GENERAL INVOLVES THE MUSCULAR CONTROL REQUIRED AROUND THE LUMBAR SPINE TO MAINTAIN FUNCTIONAL STABILITY. STABILITY AND MOVEMENT ARE CRITICALLY DEPENDENT ON THE COORDINATION OF ALL THE MUSCLES SURROUNDING THE LUMBAR SPINE. THIS PAPER AIMS TO SHOW THAT AN AGE-OLD YOGA PRACTICE, CALLED UDDHYANA BHANDA AND NOULI, IS AN EFFECTIVE MEANS OF CORE STABILITY. 2009 18 43 14 A CASE REPORT OF PATIENT PRACTISING YOGA LEADING TO DENTAL EROSION. THE ARTICLE PRESENTS THE CASE OF A PATIENT WHO WAS PRACTISING YOGA (KUNJAL KRIYA) WHICH LED TO DENTAL EROSION. DENTAL EROSION CAN BE DUE TO EXTRINSIC OR INTRINSIC CAUSES. THE INTRINSIC CAUSES INCLUDE VOMITING DUE TO ANOREXIA NERVOSA, REGURGITATION DUE TO ABNORMALITY IN GASTRO-INTESTINAL TRACT OR RUMINATION. A 38-YEAR-OLD MALE PATIENT PRESENTED WITH A RARE AETIOLOGY OF DENTAL EROSION. HE HAD PRACTICED KUNJAL KRIYA ONE OF THE YOGIC EXERCISES DESCRIBED IN ANCIENT INDIA. IN KUNJAL KRIYA THE PATIENT VOMITS ON AN EMPTY STOMACH IN ORDER TO CLEAN HIS OR HER GASTRO-INTESTINAL TRACT. THE PATIENT HAD PRACTICED THIS FORM OF EXERCISE FOR OVER 12 YEARS WHICH HAD LED TO SEVERE DENTAL EROSION. A PROPER CASE HISTORY SHOULD BE EVALUATED FOR EVERY PATIENT SO THAT THEY CAN BE COUNSELLED FOR ANY FACTORS THAT COULD BE DETRIMENTAL TO DENTAL HEALTH. EARLY DIAGNOSIS IS PARAMOUNT IN RECOGNISING THE AETIOLOGY OF DENTAL EROSION SO THAT DETRIMENTAL EFFECTS ON THE DENTITION CAN BE PREVENTED. 2007 19 2887 18 YOGA: CAN IT BE INTEGRATED WITH TREATMENT OF NEUROPATHIC PAIN? BACKGROUND: NEUROPATHIC PAIN (NP) IS A DEBILITATING CONDITION THAT MAY RESULT FROM SPINAL CORD INJURY (SCI). NEARLY 75% OF ALL SCI RESULTS IN NP AFFECTING 17,000 NEW INDIVIDUALS IN THE UNITED STATES EVERY YEAR, AND AN ESTIMATED 7-10% OF PEOPLE WORLDWIDE. IT IS CAUSED BY DAMAGED OR DYSFUNCTIONAL NERVE FIBERS SENDING ABERRANT SIGNALS TO PAIN CENTERS IN THE CENTRAL NERVOUS SYSTEM CAUSING SEVERE PAIN THAT AFFECTS DAILY LIFE AND ROUTINE. THE MECHANISMS UNDERLYING NP ARE NOT FULLY UNDERSTOOD, MAKING TREATMENT DIFFICULT. IDENTIFICATION OF SPECIFIC MOLECULAR PATHWAYS THAT ARE INVOLVED IN PAIN SYNDROMES AND FINDING EFFECTIVE TREATMENTS HAS BECOME A MAJOR PRIORITY IN CURRENT SCI RESEARCH. YOGA HAS THERAPEUTIC APPLICATIONS MAY PROVE BENEFICIAL IN TREATING SUBJECTS SUFFERING CHRONICALLY WITH SCI INDUCED NP, CHRONIC BACK AND ASSOCIATED PAINS IF NECESSARY EXPERIMENTAL DATA IS GENERATED. SUMMARY: THIS REVIEW AIMS TO DISCUSS THE IMPLICATIONS OF VARIOUS MECHANISTIC APPROACHES OF YOGA WHICH CAN BE TESTED BY NEW STUDY DESIGNS AROUND VARIOUS NOCICEPTIVE MOLECULES INCLUDING MATRIX METALLOPROTEINASES (MMPS), CATION-DEPENDENT CHLORIDE TRANSPORTER (NKCC1) ETC IN SCI INDUCED NP PATIENTS. KEY MESSAGES: THUS, YOGIC PRACTICES COULD BE USED IN MANAGING SCI INDUCED NP PAIN BY REGULATING THE ACTION OF VARIOUS MECHANISMS AND ITS ASSOCIATED MOLECULES. MODERN PRESCRIPTIVE TREATMENT STRATEGIES COMBINED WITH ALTERNATIVE APPROACHES LIKE YOGA SHOULD BE USED IN REHABILITATION CENTERS AND CLINICS IN ORDER TO AMELIORATE CHRONIC NP. WE RECOMMEND PRACTICAL CONSIDERATIONS OF CAREFUL YOGA PRACTICE AS PART OF AN INTEGRATIVE MEDICINE APPROACH FOR NP ASSOCIATED WITH SCI. 2019 20 82 15 A MECHANISTIC MODEL FOR YOGA AS A PREVENTIVE AND THERAPEUTIC MODALITY. YOGA IS AN ANCIENT INDIAN TECHNIQUE OF HEALTHY LIVING. NUMEROUS STUDIES HAVE CORROBORATED YOGA'S BENEFICIAL EFFECTS, INCLUDING A FAVORABLE INFLUENCE ON AUTONOMIC FUNCTION AND NEGATIVE EMOTIONS. EXTENSIVE RESEARCH IN THE LAST FEW DECADES HAS REVEALED THE CRITICAL ROLE THAT YOGA CAN PLAY IN ERADICATING STRESS. THIS HAS LAID TO THE FOUNDATION FOR A SCIENTIFIC UNDERSTANDING OF PATHOPHYSIOLOGICAL CHANGES ATTRIBUTED TO STRESS, PARTICULARLY AT THE MOLECULAR AND GENETIC LEVELS. THIS PRIMARILY HAS HELPED UNDERSTAND THE EPIGENETIC AND GENETIC MECHANISM AT PLAY TO INDUCE AND ALLEVIATE STRESS, PARTICULARLY THOSE RELATED TO EMOTIONAL ABERRATIONS. AS RESEARCH HAS INDICATED, NEGATIVE EMOTIONS ARE TRANSLATED INTO VASCULAR INFLAMMATION APPROPRIATELY ACCENTUATED BY A SYMPATHETIC PREDOMINANT AUTONOMIC FUNCTION. THIS CASCADE IS BOLSTERED BY MULTIPLE FACTORS, INCLUDING ACTIVATION OF "STRESSOR" GENES AND ELABORATING HORMONES, INCLUDING STEROIDS WITH SOMETIMES NOCUOUS CONSEQUENCES, PARTICULARLY WHEN CHRONIC. YOGA HAS BEEN CATEGORICALLY FOUND TO HAVE INHIBITED EACH AND EVERY ONE OF THESE BANEFUL EFFECTS OF STRESS. IN FACT, IT ALSO CHANGES THE NEURONAL CIRCUITS THAT POTENTIATE SUCH A PLETHORA OF PATHOLOGICAL CHANGES. THIS, IN TURN, HAS ACCENTUATED YOGA'S RELEVANCE AS A POWERFUL PREVENTIVE INTERVENTION IN NONCOMMUNICABLE DISEASES (NCD). NCDS, INCLUDING HEART DISEASE, STROKE, AND RHEUMATOLOGICAL DISORDERS, ARE ESSENTIALLY INFLAMMATORY DISEASES THAT PERPETUATE INFLAMMATION IN DIFFERENT BEDS LIKE VASCULAR OR JOINT SPACES. THE PRECISE MECHANISM BY WHICH YOGA INDUCES SUCH BENEFICIAL CHANGES IS YET TO BE DELINEATED. HOWEVER, A CORNUCOPIA OF POINTERS INDICATES THAT NEURAL, ENDOCRINE, IMMUNOLOGICAL, CELLULAR, GENETIC, AND EPIGENETIC MECHANISMS ARE AT PLAY. THIS ARTICLE ATTEMPTS TO COBBLE TOGETHER NEWFANGLED RESEARCH TO DELINEATE A MEDICAL MODEL FOR THIS 5000-YEAR-OLD PRACTICE FROM INDIA. THIS IS IMPERATIVE, AS A MECHANISTIC MODEL OF THIS ANCIENT-BUT-COMPLEX SYSTEM WOULD ENABLE A MORE COMPREHENSIVE UNDERSTANDING OF ITS MECHANISM AND REVEAL ITS YET-UNDISCOVERED POSITIVE HEALTH EFFECTS. 2021